2. What is ocular trauma?
Damage or trauma inflicted to the eye by external means. The
concept includes both surface injuries and intraocular injuries.
During trauma soft tissues and bony structures around the eye
maybe involved.
3. • Ocular injuries are classified as open globe, closed globe, and
periocular .
• Open globe — Open globe injuries have a full thickness break of the
eye wall, which is composed of the sclera and the cornea.
• These injuries are further described as follows:
• ●Open globe ruptures – Full thickness eye injuries caused by blunt
trauma
• ●Open globe lacerations – Full thickness eye injuries caused by
sharp objects
Closed globe injuries
• Closed globe — Closed globe injuries do not have full thickness
breaks of the eye wall. They are further divided into lamellar
lacerations (partial thickness wound to the eye wall) or
contusions (no eye wall wound).
4. • Thus, the following injuries are also lamellar lacerations:
• ●Conjunctival laceration – Full thickness break of the
conjunctiva
• ●Partial thickness scleral laceration – Incomplete scleral break
not to the level of the choroid
• ●Partial thickness corneal laceration – Incomplete corneal
break without loss of aqueous humor
• Other closed globe injuries include:
• ●Conjunctival abrasion – Injury to the epithelium of the conjunctiva
• ●Corneal abrasion – Injury to the epithelium of the cornea
• ●Hyphema – Blood in the anterior chamber of the eye
5. • ●Traumatic iritis – Inflammation in the anterior chamber resulting from
trauma
• ●Traumatic mydriasis – Chronic pupil dilation usually from iris sphincter
damage
• ●Lens dislocation – Native or artificial lens implant displacement from its
original location
• ●Vitreous hemorrhage – Bleeding into the vitreous cavity
• ●Commotio retinae – Retinal whitening due to trauma-associated retinal
edema
• ●Retinal detachment – Separation of the retina from the underlying choroid
and sclera
Periocular injuries
• The orbital septum divides the pre-septal tissues (eyelids, meibomian
glands, lashes, nasolacrimal system , and the orbicularis oculi muscle) from
the deeper orbital components (orbital bones, fat, extraocular muscles,
nerves, and vessels).
6. • Periocular injuries include:
• ●Pre-septal:
• •Eyelid abrasions – Superficial skin injury not requiring surgical repair
• •Eyelid lacerations – Full thickness skin injury usually requiring surgical
repair
• •Canalicular lacerations – Full thickness eyelid skin injury, which includes
the lacrimal drainage system; usually requires nasolacrimal cannulation for
repair
• •Periocular ecchymoses – Skin bruising, which may indicate more serious
underlying injury
Orbital :
• •Eyelid lacerations with fat prolapse – Full thickness eyelid skin
injury with penetration through orbital septum ; requires complex
repair by ophthalmology
7. • •Eyelid lacerations with levator involvement – Full thickness
eyelid skin injury, which may lead to ptosis if not repaired; requires
ophthalmology consultation
• •Orbital fractures – Fracture of any of the bones making up the
socket and surrounding the eye
• •Extraocular muscle entrapment – Prolapse of extraocular
muscle(s) into the defect created by a fractured orbital bone; may
induce muscle ischemia requiring more urgent repair
• •Orbital compartmentsyndrome (OCS) – Elevated intraorbital pressure from
infection, bleeding or inflammation causing poor motility and possible ocular ischemia
• •Orbital foreign bodies – Any post-septal foreign body present in the eye socket but
outside the globe
• •Traumatic optic neuropathy – Acute vision loss and afferent pupil defect from
trauma-induced optic nerve damage.
8. • •Optic nerve avulsion – Acute vision loss and afferent pupil defect from traumatic
transection of the optic nerve
• •Ophthalmic artery injuries – Ocular ischemia from compression or laceration of the
CONJUCTIVAL ABRASION
• Conjunctiva has less innervation than the cornea
• Less symptomatic than corneal abrasions.
ophthalmic artery
9. • Foreign body sensation, mild pain, tearing, photophobia
• Conjunctival abrasion may be invisible without fluorescein
staining.
CONJUCTIVAL
LACERATION
• Seidel test to exclude perforation of the globe (can be negative
if a full-thickness laceration is small or has spontaneously
closed)
• Inspect the conjunctiva for a foreign body.
10. • Conjunctival foreign bodies usually can be removed with a
moistened, cotton tipped applicator .
• Erythromycin ophthalmic ointment 0.5% four times a day for 2
to 3 days
CORNEAL ABRASION
Cornea is richly innervated painful
Corneal epithelium regenerates quickly, heals
within 24 to 48 hours.
11. Causes : Contact lens wear, fingernails makeup
brushes, foreign objects blown into eyes while
driving or on windy days, or which drop into the
eye while working overhead
(construction) or under a car (mechanics).
• Symptoms : Intense pain, foreign body
sensation, photophobia, and tearing
• Inspection : conjunctival injection, tearing, and
lid swelling
• Slit lamp : flare and cells from iritis if the abrasion is large and >24 hours
old, but there is no corneal infiltrate
• The abrasion usually appears as a superficial, irregular corneal defect
appearing bright green under the cobalt bluelight after instillation of
fluorescein
12. • Key to pain control is adequate and persistent cycloplegia.
• If an abrasion is >2 mm or very painful, instill a cycloplegic
agent (cyclopentolate 1% or homatropine 5%) one drop every 6
to 8 hours
• Avoid atropine because the effect lasts for approximately 2
weeks.
13. • Patients with large abrasions or abrasions in the central visual
axis should follow up with ophthalmology in 24 hours, others
at 48 to 72 hours.
• Patching the eye does not promote healing, but some patients
feel better with the eye patched
14. CORNEAL
LACERATIONS
• Misshapen iris, macro- or microhyphema, decrease in visual acuity,
and shallow anterior chamber.
• The Seidel test will be positive.
• Causes : sharp sticks, fingernails, thorns, broken glass, or sharp
toys
• Pain out of proportion to physical findings, decrease in visual
acuity, or other unexplained ocular symptoms.
15. • CT of the orbit to identify changes in globe anatomy or contour or a
foreign body within the globe, and consult ophthalmology.
ULTRAVIOLET KERATITIS
Light in the UV range can cause death of corneal epithelial cells. Classically described as “snow
blindness”
Unprotected exposure to arc welders and tanning beds, and is often called “welder’s flash.”
Blepharospasm, conjunctival injection, and prominent tearing.
Slit lamp : diffuse punctate corneal edema, and instillation of fluorescein reveals diffuse punctate
corneal abrasions.
Treatment may include double patching of both eyes if the patient requests, use of cycloplegics,
topical antibiotics (erythromycin), and oral analgesics. Healing occurs in 24 to 36 hours
16. CORNEAL FOREIGN
BODIES
• Penetration of a foreign body into the globe
can cause loss of vision.
• Metallic foreign bodies present on the cornea for more than a few hours
will cause rust to diffuse into the cornea, termed a “rust ring.”
• Inflammatory reaction, dilating blood vessels of the conjunctiva and
causing edema of the lids, conjunctiva, and cornea
• Tearing, blurred vision, and photophobia are common
• The presence of hyphema (or microhyphema in the anterior chamber on
slit lamp examination) suggests globe perforation
17. FOREIGN BODY
REMOVAL
• Irrigate with normal
saline (NS) first
• Local anesthetic such as 0.5% proparacaine
• Full-thickness corneal foreign bodies should be removed by
an ophthalmologist
• For superficial foreign bodies, a 25-gauge needle (bevel up) or
a sterile foreign body spud on an Alger brush (a low-speed,
low-torque, battery operated hand-held drill) can be used
• If a rust ring is present, the spud or an ophthalmic burr can
remove superficial rust
18. LID LACERATIONS
• Eyelid lacerations that involve the lid margin
• those within 6 to 8 mm of the medial canthus
• involving the lacrimal duct or sac,
• those involving the inner surface of the lid,
• those involving the lid margins
• wounds associated with ptosis
• those involving the tarsal plate or levator palpebrae muscle
need repair by an oculoplastic specialist.
19. • Deep lacerations medial to the punctum
potentially can transect the canalicular
system
• Instillation of fluorescein dye in the eye
with subsequent appearance in the wound
indicates loss of canalic ular integrity.
• Canalicular laceration : Repair and
Silastic tube stenting within 24 to 36
hours
• Oral cephalexin (Keflex R), 500 mg twice
or four times daily, and topical
erythromycin ophthalmic ointment four times daily .
20. • Very small lacerations (<1 mm) at the lid edge only do not need
suturing and can heal spontaneously.
• Consider the possibility of corneal laceration and globe rupture
in all full-thickness lid lacerations.
• Partial-thickness lid lacerations usually be repaired in the ED, with
referral for ophthalmologic evaluation in 2 to 3 days.
• Use a soft, absorbable or nonabsorbable 6-0 or 7-0 suture.
• Have the suture ends closest to the cornea tucked under more distant
sutures to avoid corneal irritation
21. BLUNT EYE TRAUMA
• Assessment of the visual acuity, anterior chamber, and integrity of the globe.
• If the anterior chamber is flat, a ruptured globe is certain
• Restricted upgaze or lateral gaze suggests a blow-out fracture with entrapment
and a CT scan of facial bones should be obtained.
• Feel the orbital rim above and below for step-off deformities.
• Test for cutaneous sensation along the distribution of the inferior orbital nerve
(below the eye and ipsilateral side of the nose).
• Perform a slit lamp examination with fluorescein staining to check for
abrasions, lacerations, foreign bodies, hyphema, iritis, and lens dislocation.
• If vision and ocular anatomy and function are preserved, outpatient
follow-up by an ophthalmologist in the next 48 hours.
22. • If a ruptured globe is suspected due to loss of visual acuity, flat
anterior chamber, obvious full-thickness laceration, or intraocular
foreign body, do not manipulate the eye or measure intraocular
pressure.
• Consult ophthalmology immediately.
23.
24.
25. HYPHEMA
• Blood or blood clotsin the anterior chamber are referred to as a
hyphema
• Traumatic hyphema usually: bleedingfrom a ruptured iris root vessel.
• Spontaneoushyphemas : sickle cell disease
• Treatment: Elevate the patient’s head to promote settling of suspended
RBCs inferiorly to prevent occlusion of the trabecularmeshwork.
• After consultationwith the ophthalmologist, dilate the pupil to avoid
“pupillaryplay” (constriction and dilation movements of the iris in
26. response to changing lighting conditions), which can stretch the
involved iris vessel, causing additionalbleeding
• Control of intraocular pressure consists of topical β-
blockers, IV mannitol, topical α-adrenergic agonists
(apraclonidine), and oral, topical, or IV carbonic anhydrase
inhibitors (CAIs) such as Diamox.
27. • Do not give CAIs to patients with sickle cell disease.
• patients with hyphemas occupying one third or less of the
anterior chamber can be followed closely as outpatients
28.
29. BLOW-OUT
FRACTURES
• Most frequent sites : inferior
wall (maxillary sinus) and medial wall (ethmoid sinus through
the lamina papyracea).
• Fractures of the inferior wall with entrapment of the inferior
rectus muscle can cause restriction of upgaze and diplopia
• Isolated blow-out fractures with or without entrapment can be
referred to ophthalmology, plastic surgery, OMFS , ENT for
repair within the next 3 to 10 days.
30. • A Waters view x-ray : cloudy
maxillary sinus representing blood,
fluid, and tissue on the side of the
trauma.
• Oral antibiotics (cephalexin, 250 to
500 milligrams PO four times daily
for 10 days).
• Refer to an ophthalmologist for
an outpatient full dilated
examination to rule out any
unidentified retinal tears or
detachments.
31. RUPTURED GLOBE
• Mechanism of Injury Scleral rupture may
occur from blunt or penetrating trauma
• Penetrating trauma may occur from bullets, BB pellets, knives,
sticks, darts, needles, hammering, and lawn mower projectiles.
• Suspect globe penetration with any puncture or laceration of the
eyelid or periorbital area.
• Corneal abrasions occurring when hammering metal on metal,
associated with use of high-speed machinery such as lawn mowers,
line trimmers (weed whackers), grinders, or drills, and sustained
during explosions should always be investigated for occult globe
penetration.
32. • Whenever
globe rupture is
suspected,
cover the eye with
a metal eye
shield and consult
ophthalmology immediately
• Clinical features : Decreased visual acuity, an irregular or teardrop-shaped pupil,
an afferent pupillary defect, shallow anterior chamber, hyphema, positive Seidel
test, and lens dislocation
• Presence of a large subconjunctival hemorrhage
involving the entire sclera or hemorrhagic chemosis
(bullous, raised subconjunctival hemorrhage) is very
suspicious for rupture of the globe .
• Uveal prolapse through a scleral wound may appear
as a brownish-black discoloration against the white
sclera
33. • US as well as both direct and indirect ophthalmoscopy helpful
in locating orbital and intraocular foreign bodies.
• Immediate ophthalmologic consultation
• Broad-spectrum IV antibiotics and TT administered
• Provide sedation and analgesia
• Administer antiemetic agents to prevent increased IOP and
extrusion of intraocular contents from vomiting or retching.
• The patient should be maintained NPO, anticipating surgery
34. RETROBULBAR HEMATOMA
• Severe blunt trauma to the orbit can occasionally cause a retrobulbar
hematoma.
• Abrupt increase in IOP cause decreased blood flow to the optic nerve
and loss of vision.
• Pain, proptosis, and decreasing vision.
• A CT scan of the orbit will demonstrate retrobulbar hemorrhage.
• An intraocular pressure >40 mm Hg is a consideration for emergency
canthotomy.
35. CHEMICAL
OCULAR
INJURY
• Complications of chemical burns to the eye include scarring of
the cornea with permanent loss of vision and loss of the eye due
to corneal perforation.
• Irrigation of the eyes with 1 to 2 L of NS must be done
immediately and before any examination, including testing of
vision.
36.
37. ALKALI AND ACID INJURIES
• The most serious alkali injuries are associated with
ammonia found in many household cleaners, and
lye, a common ingredient in drain cleaners.
• Lye is also a component of concrete.
• Alkali injuries cause a liquefaction necrosis,
characterized by denaturing of proteins and
saponification of fats, allowing deep penetration
into tissue.
• Acid causes coagulation necrosis, with denaturing
of protein forming a coagulum that acts as a barrier
to further tissue penetration
38. • Irrigation should be with sterile NS or
other isotonic solution and may be
instilled into the eye via a Morgan Lens
• If the pH is >7.4, continue irrigation
until the pH remains neutral 30
minutes after the last irrigation
• Conjunctival injection and chemosis, scleral whitening,
secondary to ischemia and blood vessel injury
• Patients with chemosis (edema of the bulbar conjunctiva
overlying the white sclera) and no corneal or anterior
39. chamber findings should be treated after irrigation with
erythromycin ointment four times daily
• Refer for an ophthalmologic examination in 24 to 48 hours.
• These patients are considered to have “chemical
conjunctivitis.”
• A topical cycloplegic agent should be used three times daily
for pain reduction if an epithelial defect is present.
40. CYANOACRYLATE (SUPER
GLUE/CRAZY GLUE)
• Accidental instillation into the eye and adnexa can cause
adherence of the lids and clumps of adhesive to form on the
cornea.
• To remove crazy glue, instill generous amounts of
erythromycin ointment onto the eye and on the surface of the
eyelids to moisten, lubricate, and provide antibiotic coverage
41. • The glue will loosen and become easier to remove in a few
days.
• Refer to an ophthalmologist within 24 hours for complete
removal.
