CN102723033A - Model for demonstrating heart dilation caused by excessive pressure load - Google Patents
Model for demonstrating heart dilation caused by excessive pressure load Download PDFInfo
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- CN102723033A CN102723033A CN201210207498XA CN201210207498A CN102723033A CN 102723033 A CN102723033 A CN 102723033A CN 201210207498X A CN201210207498X A CN 201210207498XA CN 201210207498 A CN201210207498 A CN 201210207498A CN 102723033 A CN102723033 A CN 102723033A
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- pulmonary artery
- sustainer
- right ventricle
- left ventricle
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Abstract
The invention relates to a model for demonstrating heart dilation caused by excessive pressure load, which relates to a teaching aid for medical teaching, and is mainly formed by an aorta model, a pulmonary artery model, an aorta outflow tract model, a pulmonary artery outflow tract model, a left ventricle model, a right ventricle model, a valvula bicuspidalis model, a tricuspid valve model and an interventricular septum model. The model is characterized in that the aorta model is communicated with the aorta outflow tract model and is finally communicated to the left ventricle model; the left ventricle model is formed by sealing a left ventricle wall model, the interventricular septum model and the valvula bicuspidalis model; the pulmonary artery model is communicated with the pulmonary artery outflow tract model and is finally communicated to the right ventricle model; the right ventricle model is formed by sealing a right ventricle wall model, the interventricular septum model and the tricuspid valve model; and an inflatable ball is respectively connected with openings of the aorta model and the pulmonary artery model. The model is appropriately used as an ocular demonstration teaching aid for explaining hypertensive disease and chronic pulmonary artery hyperbarism in majors of clinical medicine, nursing, anesthesia, ICU (Intensive Care Unit) and the like in higher medical schools.
Description
Technical field
The present invention relates to a kind of medical teaching and use teaching aid, particularly the overweight cardiac enlargement demonstrating model that causes of pressure load.
Background technology
In the medical pathologies teaching process; When telling about " hypertensive cardiopathy " and " cor pulmonale "; Though some complementary teaching aids that have sample, teaching material picture and the teaching of many bodies to provide; But do not have dynamic demonstration technical ability,, can not see the visual stimulus effect that is occurred in kind with observing though multimedia can provide a small amount of animation demonstration.
Summary of the invention
Content of the present invention:
The objective of the invention is to the deficiency that has the demonstrating model teaching aid that lacks " the overweight cardiac enlargement that causes of pressure load " now, provide a kind of lively, vivid long-term hypertension of probing into to cause that left ventricle dilatation and pulmonary hypertension cause the illustrative model of right ventricle expansion.
Realize that technical scheme of the present invention is: the overweight cardiac enlargement demonstrating model that causes of a kind of pressure load; Mainly form by sustainer model, pulmonary artery model, sustainer efferent tract model, pulmonary artery efferent tract model, left ventricle model, right ventricle model, bicuspid valve model, tricuspid valve model and interventricular septum model; It is characterized in that: sustainer model and sustainer efferent tract are connected; Lead to the left ventricle model at last, the left ventricle model is formed by left ventricular wall model, interventricular septum model and bicuspid valve model encapsulates; Pulmonary artery model and pulmonary artery efferent tract are connected, and lead to the right ventricle model at last, and the right ventricle model is formed by right ventricle wall model, interventricular septum model and tricuspid valve model encapsulates, and inflatable ball connects sustainer model and pulmonary artery model opening respectively.Said sustainer model and pulmonary artery model opening part are equipped with breather cheek valve.Cardiac module is inflatable elastomeric material.
The present invention can visual pattern demonstration go out hypertension and cause that left ventricle dilatation and pulmonary hypertension cause the pathologic process of right ventricle expansion, thereby can excite student's learning interest, stay deep study impression.
Description of drawings:
Fig. 1 is a structural representation of the present invention.
Among the figure,
The 1-inflatable ball
2-sustainer opening
3-sustainer model
4-atrium dextrum model
5-pulmonary artery model
6-pulmonary artery efferent tract model
7-tricuspid valve model
8-pulmonary artery efferent tract opening
9 right ventricle models
10-pulmonary artery opening
11-atrium sinistrum model
12-sustainer efferent tract model
13-mitral valve model
14-sustainer efferent tract opening
15-left ventricle model
16-left ventricular wall model
17-right ventricle wall model
18-interventricular septum model
The unidirectional gas admission valve of 19-.
Embodiment:
The specific embodiment of the present invention that provides through Figure of description 1 below further describes its structure; Accompanying drawing 1 is structural representation of the present invention; Referring to accompanying drawing 1; The overweight cardiac enlargement demonstrating model that causes of pressure load; Mainly form by sustainer model 3, pulmonary artery model 5, sustainer efferent tract model 12, pulmonary artery efferent tract model 6, left ventricle model 15, right ventricle model 9, bicuspid valve model 13, tricuspid valve model 7 and interventricular septum model 18; Sustainer model 3 is connected with sustainer efferent tract model 12, leads to left ventricle model 15 at last through sustainer efferent tract opening 14, and left ventricle model 15 is formed by left ventricular wall model 16, interventricular septum model 18 and 13 sealings of bicuspid valve model; Pulmonary artery model 5 is connected with pulmonary artery efferent tract model 6; Lead to right ventricle model 9 at last through pulmonary artery efferent tract opening 8; Right ventricle model 9 is formed by right ventricle wall model 17, interventricular septum model 18 and 7 sealings of tricuspid valve model, and inflatable ball 1 connects sustainer model opening 2 and pulmonary artery model opening 10 respectively.Said sustainer model and pulmonary artery model opening are equipped with breather cheek valve 19.Cardiac module as shown in the figure, as to the present invention relates in order to increase and the anatomical cardiac similarity, is provided with atrium sinistrum model 11 and atrium dextrum model 4 above cardiac module.
During use; When simulation hypertension; Inflatable ball 1 pressurization inflation makes air pass through unidirectional gas admission valve 19 and imports sustainer model 3, goes forward side by side into sustainer efferent tract model 12; Arrive left ventricle model 15 through sustainer efferent tract opening 14, thereby simulation ejection impedance increase causing left ventricle bears bigger pressure load and makes 15 expansions of left ventricle model when shrinking.
When the simulated lung arterial hypertension; Through inflatable ball 1 pressurization inflation; Make air pass through unidirectional gas admission valve 19 and import pulmonary artery model 5; And get into pulmonary artery efferent tract model 6, arrive right ventricle model 9 through pulmonary artery efferent tract opening 8, thereby simulation ejection impedance increase causing right ventricle bears bigger pressure load and makes 9 expansions of right ventricle model when shrinking.
From the above, the present invention can be intuitively, demonstrate out the overweight pathologic process that causes cardiac enlargement of pressure load visually.
Claims (2)
1. the overweight cardiac enlargement demonstrating model that causes of pressure load; Mainly form by sustainer model, pulmonary artery model, sustainer efferent tract model, pulmonary artery efferent tract model, left ventricle model, right ventricle model, bicuspid valve model, tricuspid valve model and interventricular septum model; It is characterized in that: sustainer model and sustainer efferent tract are connected; Lead to the left ventricle model at last, the left ventricle model is formed by left ventricular wall model, interventricular septum model and bicuspid valve model encapsulates; Pulmonary artery model and pulmonary artery efferent tract are connected, and lead to the right ventricle model at last, and the right ventricle model is formed by right ventricle wall model, interventricular septum model and tricuspid valve model encapsulates, and inflatable ball connects sustainer model and pulmonary artery model opening respectively.
2. according to claim 1, the overweight cardiac enlargement demonstrating model that causes of described pressure load is characterized in that said sustainer model and pulmonary artery model opening part are equipped with breather cheek valve.
Priority Applications (1)
| Application Number | Priority Date | Filing Date | Title |
|---|---|---|---|
| CN201210207498XA CN102723033A (en) | 2012-06-22 | 2012-06-22 | Model for demonstrating heart dilation caused by excessive pressure load |
Applications Claiming Priority (1)
| Application Number | Priority Date | Filing Date | Title |
|---|---|---|---|
| CN201210207498XA CN102723033A (en) | 2012-06-22 | 2012-06-22 | Model for demonstrating heart dilation caused by excessive pressure load |
Publications (1)
| Publication Number | Publication Date |
|---|---|
| CN102723033A true CN102723033A (en) | 2012-10-10 |
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Family Applications (1)
| Application Number | Title | Priority Date | Filing Date |
|---|---|---|---|
| CN201210207498XA Pending CN102723033A (en) | 2012-06-22 | 2012-06-22 | Model for demonstrating heart dilation caused by excessive pressure load |
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| CN (1) | CN102723033A (en) |
Citations (7)
| Publication number | Priority date | Publication date | Assignee | Title |
|---|---|---|---|---|
| CN2078033U (en) * | 1990-11-09 | 1991-05-29 | 温州师范学院 | Blood function demonstrator of heart pump |
| CN2293098Y (en) * | 1997-03-26 | 1998-09-30 | 温州师范学院教学仪器厂 | Heart pump blood function demonstrator |
| CN2327032Y (en) * | 1998-03-03 | 1999-06-30 | 杨喜山 | Blood-circulation analogue demonstrator |
| US6062866A (en) * | 1998-03-27 | 2000-05-16 | Prom; James M. | Medical angioplasty model |
| US20040033477A1 (en) * | 2002-04-03 | 2004-02-19 | Ramphal Paul S. | Computer-controlled tissue-based simulator for training in cardiac surgical techniques |
| CN2607635Y (en) * | 2002-12-20 | 2004-03-24 | 张导华 | Human heart model |
| CN202632623U (en) * | 2012-06-22 | 2012-12-26 | 关真民 | Demonstration model of heart dilation caused by pressure overload |
-
2012
- 2012-06-22 CN CN201210207498XA patent/CN102723033A/en active Pending
Patent Citations (7)
| Publication number | Priority date | Publication date | Assignee | Title |
|---|---|---|---|---|
| CN2078033U (en) * | 1990-11-09 | 1991-05-29 | 温州师范学院 | Blood function demonstrator of heart pump |
| CN2293098Y (en) * | 1997-03-26 | 1998-09-30 | 温州师范学院教学仪器厂 | Heart pump blood function demonstrator |
| CN2327032Y (en) * | 1998-03-03 | 1999-06-30 | 杨喜山 | Blood-circulation analogue demonstrator |
| US6062866A (en) * | 1998-03-27 | 2000-05-16 | Prom; James M. | Medical angioplasty model |
| US20040033477A1 (en) * | 2002-04-03 | 2004-02-19 | Ramphal Paul S. | Computer-controlled tissue-based simulator for training in cardiac surgical techniques |
| CN2607635Y (en) * | 2002-12-20 | 2004-03-24 | 张导华 | Human heart model |
| CN202632623U (en) * | 2012-06-22 | 2012-12-26 | 关真民 | Demonstration model of heart dilation caused by pressure overload |
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Application publication date: 20121010 |